Özdemir D, et al. - Because vitamin B12 deficiency-induced hyperhomocysteinemia was associated with impaired microarterial flow, demyelization, and neuronal damage, resulting in cochlear damage and auditory dysfunction, researchers assessed the possible vestibular-evoked myogenic potential (VEMP) abnormalities in vitamin B12 deficiency patients. For this prospective study, they compared 37 patients diagnosed with vitamin B12 deficiency (< 220 pg/mL) with 31 audiologically healthy candidates with normal B12 levels. In the patient group, the rate of absent VEMP responses was twice as high as in the healthy control group. In addition, the mean interpeak amplitude values in both right and left ears in the patient group were statistically shorter than the control group. Similarly, the mean amplitude asymmetry ratio in the patient group was statistically higher than the control group. There have been no statistically significant differences between groups in latency responses. In patients with vitamin B12 deficiency, increased rates of absent VEMPs and reduced amplitudes with normal latencies are attributed to peripheral vestibular hypofunction.