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Reproductive and metabolic determinants of granulosa cell dysfunction in normal-weight women with polycystic ovary syndrome

Fertility and Sterility Feb 15, 2018

Guedikian AA, et al. - Researchers aimed at determining the degree to which E2 hyperresponsiveness to FSH and antimüllerian hormone (AMH) overproduction in normal-weight women with polycystic ovary syndrome (PCOS) correlate with increased antral follicle number (AFN), hyperandrogenism, and/or metabolic dysfunction. Findings revealed that in normal-weight women with PCOS, serum E2 hyperresponsiveness to recombinant human (rh) FSH represented increased AFN, while elevated serum AMH levels reflected opposing effects of stimulatory reproductive (hyperandrogenism and increased AFN) vs inhibitory metabolic (body fat) factors.
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